Periodontal Disease Ebooks Catalog

Freedom From Dental Disease

The Primary Care Oral Health Action Pack can Favorably change the way you look at your oral health and what affects it. Increase your knowledge of YOUR teeth and gums, YOUR entire oral environment and yes, your SMILE! Reveal the TRUE CAUSE of cavities, decay of bone and tissue and disclose the proper biological balance. Put YOU in command over this contagious, yet EASILY preventable disease which runs rampant thoughout our population. Allow you to TAKE ACTION to restoring your teeth and gums to optimum health while preventing further decay and damage, as well as preventing any potentially related problems such as heart disease, diabetes, and so on. How to Become Dentally Self Sufficient. Research Advocates OraMedics The science behind the program! The 7 Factors Transcript - Dr. Nara's Last speech before the World Health Federation. Continue reading...

Freedom From Dental Disease Summary

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Official Website: mizar5.com
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What You Should Know About Gum Disease

These books encapsulate my personal experience with fighting gum disease, described as follows: My gums bled during dental cleanings. I had pockets depths of 4 and 5mms and one 6mm pocket. My dentist told me I had moderate gum (periodontal) disease. She and / or the hygienist also told me that the ONLY thing that would help me was a Scaling and Root Planing Treatment - Otherwise knowns as a SRP or Deep Cleaning. After applying the information I learned on my own and the tools that I employed the results were that my pocket depths returned to normal. There was no more bleeding during dental cleanings, brushing or flossing. I was told that I no longer needed the Deep Cleaning or SRP treatment and that whatever I was doing, I should keep it up. Continue reading...

What You Should Know About Gum Disease Summary

Format: Ebook
Official Website: gingivitiskiller.com
Price: $19.65

Tolllike receptors and atherosclerosis Genetic evidence

Several polymorphisms in the genes encoding TLRs have been investigated in the context of infectious diseases 79 , autoimmune disorders 80 , allergies 81 , periodontal disease 82 , and renal disorders 83 , but have also focused on a potential role of TLR polymorphisms on vascular disease and its clinical sequelae. Patients either heterozygous or homozygous for two different single nucleotide polymorphisms (Asp299Gly and Thr399Ile) that map to the extracellular domain of TLR4 are hyporesponsive to a challenge with LPS 84 . Atherosclerosis is characterized by chronic local inflammation 5, 85 , and thus a blunted response to LPS might place the host at a disadvantage in eradicating invading microorganisms yet could diminish cardiovascular risk because of decreased systemic inflammation. Therefore, patients harboring Asp299Gly and Thr399Ile polymorphisms might be imbued with greater cardiovascular protection. A population-based epidemiologic study did indeed show that subjects carrying...

Perinatal transmission See transmission

Periodontal disease Disease of supporting structures of the teeth, the periodontium, including the alveolar bone to which the teeth are anchored. The most common symptom is bleeding gums, but loosening of the teeth, receding gums and teeth, and necrotizing ulcerative gingivitis may be present as the process continues. Proper dental hygiene will help to prevent peri-odontal disease.

The Line That Cant Be Crossed

In some cases, tissues in the body simply need to be making more of something they already make. The item to be supplemented is not missing and the gene is not mutated. One of the situations in which this approach is being used is to get cells to make the proteins necessary for the formation of new bone material (Figure 35.4). In these cases, the patient does not have a defect in bone formation but rather has an injury of some kind that is more than his own body can heal. Gene therapy treatment of skin cells with bone mor-phogenic protein before placement of the cells into a region of bone erosion in periodontal disease can lead to formation of new bone in the region. Another approach places the gene therapy agents into a gel placed at the

Baby with Petechiae and Bruises

This baby received his subsequent childhood immunizations subcutaneously, rather than intramuscularly, in order to prevent intramuscular hematoma. His parents made sure that he had soft toys to play with and padded sharp corners and ledges at home. When his mobility increases over the next few months, he will be fitted with a soft helmet. Soft helmets may prevent or lessen head trauma in toddlers and children, but many families and older patients dislike the stigmatization. Dental hygiene practices are especially important in patients with bleeding tendencies, both because gingivitis is associated with bleeding, and also because tooth extraction is associated with risk of hematoma and bleeding.

Biofilms in medical systems

Surfaces within the mouth become readily colonized with bacterial deposits, forming a biofilm, usually called dental plaque. By attaching to the teeth or dental implants, the biofilm helps to prevent colonization of the mouth by pathogenic bacteria. Although dental plaque forms naturally without good oral hygiene, it can be a source of dental caries or periodontal disease. The attached organisms obtain nutrients from the ingested food, saliva, and gingival crevice fluid found between the teeth and gums It is thought that most of the nutrients are derived from the host rather than the from the host's diet. Dental caries are formed as a result of the localized dissolution of the tooth enamel by acids produced by metabolism of carbohydrates, lowering the pH and favoring the growth of mutans streptococci and lacto-bacilli. Periodontal diseases occur when the supporting tissues of the teeth are attacked by obligately anaerobic gram-negative rods, filaments, or spiral-shaped bacteria....

Oral Microbial Ecology

The characteristics and properties of the microflora at various sites in the mouth, like those of any microbial ecosystem, are regulated by the nature of the habitat and the associated environment. In this respect, some discussion of the microbial ecology of the mouth is important, particularly since the two major diseases of dental caries and periodontal disease arise through alterations of the normal, or indigenous, flora rather than the invasion of foreign or alien infectious agents from outside the mouth. Failure to remove the bacteria at the gingival margin can lead to inflammation (gingivitis) and eventual destruction of the soft tissues next to the teeth, creating a pocket containing a largely Gram-negative anaerobic flora with a considerable capacity for proteolysis. Individuals with such pockets have periodontal disease and the ultimate stage of the disease is known as destructive periodontitis. This condition is characterized by the presence of large pockets 8-10 mm in depth...

Wegeners granulomatosis

Mucosal manifestations might also be suggestive of WG. Buccal lesions are undoubtedly frequent, often reported as one of the ear, nose and throat manifestations of WG, present in 10-50 of the patients (D'Cruz et al., 1989 Frances et al., 1994). Their numbers and localizations vary widely, and, unlike recurrent aphthae, they are persistent. Hyperplastic gingivitis and gums are rare, but with some well-documented case reports (Patten and Tomecki, 1993). The gingiva is generally described as granular and red to purple with many petechiae. The entire gingiva and periodontium may be involved resulting in tooth mobility and loss. Major but incomplete regression can be obtained with empiric antimicrobial therapy. Genital ulcers are

Cohort studies 111

Is found in mammalian tissue, with the highest concentrations in the heart, liver, kidney, and muscle. Co-Q10 levels are abnormally low in people with congestive heart failure and in populations with HIV, muscular dystrophy, periodontal disease, immune dysfunction, and immunosuppression caused by the cancer chemotherapy doxorubicin. To date, no toxicities have been reported from Co-Q10 use.

Pathogenesis

The pathogenesis of P. gingivalis relies on a broad range of virulence factors. This organism is a prominent etiologic agent in adult periodontitis. The progression of this disease is hypothesized to be related to the production of a variety of enzymes (particularly proteolytic enzymes), fimbriae, capsular polysaccha-ride, LPS, hemagglutinin, and hemolytic activity. P. gingivalis has been shown to invade and replicate host cells, a mechanism that may facilitate its spread. A class of trypsin-like cysteine proteases, termed gingipains, have recently been implicated as a major virulence factor contributing to the tissue destruction that is the hallmark of periodontal disease. The capsular polysaccharide of P. gingivalis acts as a potent virulence factor facilitating a spreading infection in mice greater than that seen with unencapsu-lated strains. The LPS of P. gingivalis has been implicated in the initiation and development of periodontal disease. It has been shown that the LPS...

Immunity

Given live P. gingivalis yielded an increase in serum and salivary responses compared to control animals. These results indicate a role for serum IgG and salivary IgA in protection against periodontal disease in which a balance between Th1 and Th2-like cells occurs in humoral immune responses to P. gingivalis. In a recent clinical study, patients with periodontal disease develop a significant antibody response to P. gingivalis, but this response does not eliminate infection.

Normal oral flora

Comparisons made among different species of animals show that while some genera are common to several mammals, different animal species show variations in the complexity of their flora and harbour different species of a given microbial genus. Among the best-studied animals, apart from humans, are those which naturally develop caries or periodontal disease, and those in which these diseases can be produced experimentally. The oral flora of the nonhuman primates is similar to that of humans, while dogs and cats are also known to have complex micro-bial communities. The flora of other animals, such as hamsters, rats and mice, is less complex. Few studies of the oral flora in herbivores, insectivores and carnivores, other than cats and dogs, have been made and nothing is known of the microflora which is resident in the oral cavities of reptiles and fishes. Even in humans and primates, relatively few studies have It is likely that given similar environments in different animal species,...

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